Fig. 2

Comparison of membrane bound CD100 (mCD100) expression in CD4⁺ T cells and CD8⁺ T cells in the plasma among control group (n = 20), stable angina pectoris (SAP) group (n = 22), unstable angina pectoris (UAP) group (n = 20), and acute myocardial infarction (AMI) group (n = 23). a PBMCs were stained by anti-CD3, CD4, CD8, and CD100, and the representative flow dots and histograms of CD100 expression in CD4⁺ T cells and in CD8⁺ T cells in control, SAP, UAP, and AMI group were shown. The isotype control for CD100 positive and negative cells was also shown. b Comparison of CD100⁺CD4⁺ cells percentage among control, SAP, UAP, and AMI group. There was no significant difference of CD100⁺CD4⁺ cells percentage among control, SAP, UAP, and AMI group. c Comparison of CD100 mean fluorescence intensity (MFI) in CD4⁺ T cells among control, SAP, UAP, and AMI group. There was no significant difference of CD100 MFI in CD4⁺ T cells among control, SAP, UAP, and AMI group. d Comparison of CD100⁺CD8⁺ cells percentage among control, SAP, UAP, and AMI group. CD100⁺CD8⁺ cells percentage was reduced in AMI group compared with control, SAP, and UAP group. e Comparison of CD100 MFI in CD8⁺ T cells among control, SAP, UAP, and AMI group. CD100 MFI in CD8⁺ T cells was down-regulated in AMI group compared with control, SAP, and UAP group. Individual level for each subject was shown. The horizon lines indicated means, while the error bars indicated standard deviations. One-way ANOVA and SNK-q test was used for statistical analysis